Role of the E1A Rb-binding domain in repression of the NF- B-dependent defense against tumor necrosis factor-
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference80 articles.
1. Induction by E1A oncogene expression of cellular susceptibility to lysis by TNF
2. p53-dependent apoptosis modulates the cytotoxicity of anticancer agents
3. Induction of susceptibility to tumor necrosis factor by E1A is dependent on binding to either p300 or p105-Rb and induction of DNA synthesis
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