Pten heterozygosity restores neuronal morphology in fragile X syndrome mice
Author:
Affiliation:
1. Department of Molecular and Systems Biology, Geisel School of Medicine at Dartmouth, Hanover, NH 03755
2. Department of Biochemistry and Cellular Biology, Geisel School of Medicine at Dartmouth, Hanover, NH 03755
Abstract
Funder
HHS | NIH | National Institute of Mental Health
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Link
https://pnas.org/doi/pdf/10.1073/pnas.2109448119
Reference65 articles.
1. Genetic Removal of p70 S6 Kinase 1 Corrects Molecular, Synaptic, and Behavioral Phenotypes in Fragile X Syndrome Mice
2. FMRP Stalls Ribosomal Translocation on mRNAs Linked to Synaptic Function and Autism
3. The Role of PTEN in Neurodevelopment
4. Excess Phosphoinositide 3-Kinase Subunit Synthesis and Activity as a Novel Therapeutic Target in Fragile X Syndrome
5. Hyperactivity of Newborn Pten Knock-out Neurons Results from Increased Excitatory Synaptic Drive
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