Bloom's and Werner's syndrome genes suppress hyperrecombination in yeast sgs1 mutant: Implication for genomic instability in human diseases
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference30 articles.
1. Evidence for increased in vivo mutation and somatic recombination in Bloom's syndrome.
2. Clonal structural chromosomal rearrangements in primary fibroblast cultures and in lymphocytes of patients with Werner's syndrome
3. Mutator phenotype of Werner syndrome is characterized by extensive deletions.
4. Nucleotide sequence analysis of human hypoxanthine phosphoribosyltransferase (HPRT) gene deletions
5. The Bloom's syndrome gene product is homologous to RecQ helicases
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