Bcl-2 expression causes redistribution of glutathione to the nucleus
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference31 articles.
1. Bcl-2-deficient mice demonstrate fulminant lymphoid apoptosis, polycystic kidneys, and hypopigmented hair
2. Bcl-2 functions in an antioxidant pathway to prevent apoptosis
3. X-ray and NMR structure of human Bcl-xL, an inhibitor of programmed cell death
4. Bcl-xL forms an ion channel in synthetic lipid membranes
5. Resistance to radiation-induced apoptosis in Bcl-2-expressing cells is reversed by depleting cellular thiols
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