Cardiomyocyte-restricted knockout of STAT3 results in higher sensitivity to inflammation, cardiac fibrosis, and heart failure with advanced age-Reference-Cited by-同舟云学术

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Cardiomyocyte-restricted knockout of STAT3 results in higher sensitivity to inflammation, cardiac fibrosis, and heart failure with advanced age

Published:2003-10-17 Issue:22 Volume:100 Page:12929-12934
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Jacoby J. J.,Kalinowski A.,Liu M.-G.,Zhang S. S.- M.,Gao Q.,Chai G.-X.,Ji L.,Iwamoto Y.,Li E.,Schneider M.,Russell K. S.,Fu X.-Y.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference24 articles.

1. Cytokines and Cytokine Receptors in Advanced Heart Failure

2. gp130 AND THE INTERLEUKIN-6 FAMILY OF CYTOKINES

3. Targeted disruption of gp130, a common signal transducer for the interleukin 6 family of cytokines, leads to myocardial and hematological disorders.

4. Loss of a gp130 Cardiac Muscle Cell Survival Pathway Is a Critical Event in the Onset of Heart Failure during Biomechanical Stress

5. Signaling mechanisms through gp130: A model of the cytokine system

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