Pathogenetic sequence for aneurysm revealed in mice underexpressing fibrillin-1-Reference-Cited by-同舟云学术

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Pathogenetic sequence for aneurysm revealed in mice underexpressing fibrillin-1

Published:1999-03-30 Issue:7 Volume:96 Page:3819-3823
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Pereira L.,Lee S. Y.,Gayraud B.,Andrikopoulos K.,Shapiro S. D.,Bunton T.,Biery N. J.,Dietz H. C.,Sakai L. Y.,Ramirez F.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference23 articles.

1. Mutations in the human gene for fibrillin-1 (FBN1) in the Marfan syndrome and related disorders

2. Fibrillin mutations in Marfan syndrome and related phenotypes

3. Targetting of the gene encoding fibrillin–1 recapitulates the vascular aspect of Marfan syndrome

4. Targeted mutation of the DNA methyltransferase gene results in embryonic lethality

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1. Tissue material properties, whole-bone morphology and mechanical behavior in the Fbn1C1041G/+ mouse model of Marfan syndrome;Matrix Biology Plus;2024-08

2. Myeloid-Specific Thrombospondin-1 Deficiency Exacerbates Aortic Rupture via Broad Suppression of Extracellular Matrix Proteins;2024-07-31

3. Novel Insights into the Aortic Mechanical Properties of Mice Modeling Hereditary Aortic Diseases;Thrombosis and Haemostasis;2024-07-01

4. Poglut2/3 double knockout in mice results in neonatal lethality with reduced levels of fibrillin in lung tissues;Journal of Biological Chemistry;2024-07

5. The Fbn1 gene variant governs passive ascending aortic mechanics in the mgΔlpn mouse model of Marfan syndrome when superimposed to perlecan haploinsufficiency;Frontiers in Cardiovascular Medicine;2024-03-13

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