A critical role for the peroxisome proliferator-activated receptor (PPAR ) in the cellular fasting response: The PPAR -null mouse as a model of fatty acid oxidation disorders-Reference-Cited by-同舟云学术

A critical role for the peroxisome proliferator-activated receptor (PPAR ) in the cellular fasting response: The PPAR -null mouse as a model of fatty acid oxidation disorders

Published:1999-06-22 Issue:13 Volume:96 Page:7473-7478
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Leone T. C.,Weinheimer C. J.,Kelly D. P.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference39 articles.

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2. Regulation of palmitate metabolism by carnitine and glucagon in hepatocytes isolated from fasted and carbohydrate refed rats

3. Enhancement of mitochondrial carnitine and carnitine acylcarnitine translocase-mediated transport of fatty acids into liver mitochondria under ketogenic conditions.

4. Carnitine palmitoyltransferase: activation and inactivation in liver mitochondria from fed, fasted, hypo- and hyperthyroid rats

5. Developmental, nutritional, and hormonal regulation of tissue-specific expression of the genes encoding various acyl-CoA dehydrogenases and alpha-subunit of electron transfer flavoprotein in rat.

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