Defects in inositol 1,4,5-trisphosphate receptor expression, Ca2+ signaling, and insulin secretion in the anx7(+/-) knockout mouse-Reference-Cited by-同舟云学术

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Defects in inositol 1,4,5-trisphosphate receptor expression, Ca2+ signaling, and insulin secretion in the anx7(+/-) knockout mouse

Published:1999-11-23 Issue:24 Volume:96 Page:13783-13788
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Srivastava M.,Atwater I.,Glasman M.,Leighton X.,Goping G.,Caohuy H.,Miller G.,Pichel J.,Westphal H.,Mears D.,Rojas E.,Pollard H. B.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference30 articles.

1. “Annexinopathies” — A New Class of Diseases

2. Identification and purification of an adrenal medullary protein (synexin) that causes calcium-dependent aggregation of isolated chromaffin granules.

3. Annexins: the problem of assessing the biological role for a gene family of multifunctional calcium- and phospholipid-binding proteins

4. Membrane fusion protein synexin (annexin VII) as a Ca2+/GTP sensor in exocytotic secretion.

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