Aging accentuates and bone marrow transplantation ameliorates metabolic defects in Fabry disease mice-Reference-Cited by-同舟云学术

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Aging accentuates and bone marrow transplantation ameliorates metabolic defects in Fabry disease mice

Published:1999-05-25 Issue:11 Volume:96 Page:6423-6427
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Ohshima T.,Schiffmann R.,Murray G. J.,Kopp J.,Quirk J. M.,Stahl S.,Chan C.-C.,Zerfas P.,Tao-Cheng J.-H.,Ward J. M.,Brady R. O.,Kulkarni A. B.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference15 articles.

1. Enzymatic Defect in Fabry's Disease

2. Replacement Therapy for Inherited Enzyme Deficiency

3. Treatment of genetic defects in hematopoietic cell function by gene transfer

4. Gene therapy for metabolic disorders

5. -Galactosidase A deficient mice: A model of Fabry disease

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1. Comparative study on incorporation of three recombinant human α-galactosidase A drugs (agalsidases) into cultured fibroblasts and organs/tissues of Fabry mice;Molecular Genetics and Metabolism Reports;2024-09

2. Fabry Disease Rat Model Develops Age- and Sex-Dependent Anterior Segment Ocular Abnormalities;Investigative Ophthalmology & Visual Science;2024-08-07

3. Targeted gene therapy for rare genetic kidney diseases;Kidney International;2024-08

4. The role of tubular cells in the pathogenesis of Fabry nephropathy;Frontiers in Cardiovascular Medicine;2024-04-05

5. Therapeutic Strategy for Fabry Disease by Intravenous Administration of Adeno-Associated Virus 9 in a Symptomatic Mouse Model;Human Gene Therapy;2024-03-01

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