A small number of residues in the class II molecule I-Au confer the ability to bind the myelin basic protein peptide Ac1-11
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference47 articles.
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1. A previously unappreciated polymorphism in the beta chain of I-As expressed in autoimmunity-prone SJL mice: Combined impact on antibody, CD4 T cell recognition and MHC class II dimer structural stability;Molecular Immunology;2022-03
2. Death, adaptation and regulation: The three pillars of immune tolerance restrict the risk of autoimmune disease caused by molecular mimicry;Journal of Autoimmunity;2007-12
3. CD4+CD25+ regulatory T cells limit the risk of autoimmune disease arising from T cell receptor crossreactivity;Proceedings of the National Academy of Sciences;2005-11-15
4. Fas-mediated death and sensory adaptation limit the pathogenic potential of autoreactive T cells after strong antigenic stimulation;Journal of Leukocyte Biology;2005-04-13
5. Activation thresholds determine susceptibility to peptide-induced tolerance in a heterogeneous myelin-reactive T cell repertoire;Journal of Neuroimmunology;2004-11
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