Down-regulation of the expression of endothelial NO synthase is likely to contribute to glucocorticoid-mediated hypertension-Reference-Cited by-同舟云学术

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Down-regulation of the expression of endothelial NO synthase is likely to contribute to glucocorticoid-mediated hypertension

Published:1999-11-09 Issue:23 Volume:96 Page:13357-13362
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Wallerath T.,Witte K.,Schafer S. C.,Schwarz P. M.,Prellwitz W.,Wohlfart P.,Kleinert H.,Lehr H.-A.,Lemmer B.,Forstermann U.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference34 articles.

1. Role of endothelium-derived nitric oxide in the regulation of blood pressure.

2. Hypertension in mice lacking the gene for endothelial nitric oxide synthase

3. Endothelium-derived relaxing factor from cultured human endothelial cells inhibits aggregation of human platelets

4. The role of nitric oxide and cGMP in platelet adhesion to vascular endothelium

5. Leukocyte-endothelial cell adhesion in spontaneously hypertensive and normotensive rats.

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