p53 represses c-Myc through induction of the tumor suppressor miR-145
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference33 articles.
1. Complementation by wild-type p53 of interleukin-6 effects on M1 cells: induction of cell cycle exit and cooperativity with c-myc suppression.
2. The many faces of c-MYC
3. c-Myc overexpression and endocrine resistance in breast cancer
4. p53-Dependent Transcriptional Repression of c-myc Is Required for G 1 Cell Cycle Arrest
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