Frataxin activates mitochondrial energy conversion and oxidative phosphorylation-Reference-Cited by-同舟云学术

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Frataxin activates mitochondrial energy conversion and oxidative phosphorylation

Published:2000-10-17 Issue:22 Volume:97 Page:12239-12243
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Ristow M.,Pfister M. F.,Yee A. J.,Schubert M.,Michael L.,Zhang C.-Y.,Ueki K.,Michael M. D.,Lowell B. B.,Kahn C. R.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference51 articles.

1. Glucose metabolism alterations in Friedreich's ataxia

2. Friedreich's Ataxia and Oral Glucose Tolerance: I. The effect of ingested glucose on serum glucose and insulin values in homozygotes, obligate heterozygotes and potential carriers of the Friedreich's ataxia gene.

3. Glucose intolerance in Friedreich's Ataxia: Association with insulin resistance and decreased insulin binding

4. Heterozygous expansion of the GAA tract of the X25/frataxin gene is associated with insulin resistance in humans.

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1. mTORC1 Signaling Inhibition Modulates Mitochondrial Function in Frataxin Deficiency;2024-08-07

2. Interactional Analysis of Single Mitochondrial Function Directly in Living Cell Reveals the Proton Circuit Decoupling of Mitochondria at the Preliminary Stage of Apoptosis;CCS Chemistry;2024-04-11

3. Human frataxin, the Friedreich ataxia deficient protein, interacts with mitochondrial respiratory chain;Cell Death & Disease;2023-12-08

4. Novel intragenic deletion within the FXN gene in a patient with typical phenotype of Friedreich ataxia: may be more prevalent than we think?;BMC Medical Genomics;2023-12-01

5. Decoding Neurodegeneration: A Comprehensive Review of Molecular Mechanisms, Genetic Influences, and Therapeutic Innovations;International Journal of Molecular Sciences;2023-08-21

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