Aberrant S-nitrosylation mediates calcium-triggered ventricular arrhythmia in the intact heart-Reference-Cited by-同舟云学术

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Aberrant S-nitrosylation mediates calcium-triggered ventricular arrhythmia in the intact heart

Published:2012-10-15 Issue:44 Volume:109 Page:18186-18191
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Cutler M. J.,Plummer B. N.,Wan X.,Sun Q.-A.,Hess D.,Liu H.,Deschenes I.,Rosenbaum D. S.,Stamler J. S.,Laurita K. R.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference24 articles.

1. Cardiac electrophysiological effects of nitric oxide

2. S -Nitrosylation in Cardiovascular Signaling

3. Regulation of the Cardiac Muscle Ryanodine Receptor by O2 Tension and S-Nitrosoglutathione

4. A SNO Storm in Skeletal Muscle

5. Deficient ryanodine receptor S-nitrosylation increases sarcoplasmic reticulum calcium leak and arrhythmogenesis in cardiomyocytes

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1. S100A1’s single cysteine is an indispensable redox switch for the protection against diastolic calcium waves in cardiomyocytes;American Journal of Physiology-Heart and Circulatory Physiology;2024-07-01

2. Protein carbonylation causes sarcoplasmic reticulum Ca2+ overload by increasing intracellular Na+ level in ventricular myocytes;Pflügers Archiv - European Journal of Physiology;2024-05-20

3. Maternal exercise preserves offspring cardiovascular health via oxidative regulation of the ryanodine receptor;Molecular Metabolism;2024-04

4. Protein carbonylation causes sarcoplasmic reticulum Ca2+ overload by increasing intracellular Na+ level in ventricular myocytes;2024-03-01

5. S100A1’s single cysteine is an indispensable redox-switch for the protection against diastolic calcium leakage in cardiomyocytes;2023-09-01

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