Analysis of the tumor-initiating and metastatic capacity of PDX1-positive cells from the adult pancreas
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference55 articles.
1. Activated Kras and Ink4a/Arf deficiency cooperate to produce metastatic pancreatic ductal adenocarcinoma
2. Both p16Ink4a and the p19Arf-p53 pathway constrain progression of pancreatic adenocarcinoma in the mouse
3. Chronic Pancreatitis Is Essential for Induction of Pancreatic Ductal Adenocarcinoma by K-Ras Oncogenes in Adult Mice
4. Context-Dependent Transformation of Adult Pancreatic Cells by Oncogenic K-Ras
5. Metastatic Pancreatic Cancer Is Dependent on Oncogenic Kras in Mice
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