JAK/STAT1 signaling promotes HMGB1 hyperacetylation and nuclear translocation-Reference-Cited by-同舟云学术

JAK/STAT1 signaling promotes HMGB1 hyperacetylation and nuclear translocation

Published:2014-01-27 Issue:8 Volume:111 Page:3068-3073
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Lu B.,Antoine D. J.,Kwan K.,Lundback P.,Wahamaa H.,Schierbeck H.,Robinson M.,Van Zoelen M. A. D.,Yang H.,Li J.,Erlandsson-Harris H.,Chavan S. S.,Wang H.,Andersson U.,Tracey K. J.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference41 articles.

1. HMGB proteins function as universal sentinels for nucleic-acid-mediated innate immune responses

2. HMGB1 Is a Therapeutic Target for Sterile Inflammation and Infection

3. HMGB1 and leukocyte migration during trauma and sterile inflammation

4. Toll-like receptor 4 and high-mobility group box-1 are involved in ictogenesis and can be targeted to reduce seizures

5. Induction of inflammatory and immune responses by HMGB1–nucleosome complexes: implications for the pathogenesis of SLE

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