Molecular basis for chromatin binding and regulation of MLL5-Reference-Cited by-同舟云学术

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Molecular basis for chromatin binding and regulation of MLL5

Published:2013-06-24 Issue:28 Volume:110 Page:11296-11301
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Ali M.,Rincon-Arano H.,Zhao W.,Rothbart S. B.,Tong Q.,Parkhurst S. M.,Strahl B. D.,Deng L.-W.,Groudine M.,Kutateladze T. G.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference21 articles.

1. MLL5, a homolog of Drosophila trithorax located within a segment of chromosome band 7q22 implicated in myeloid leukemia

2. Loss of Mll5 results in pleiotropic hematopoietic defects, reduced neutrophil immune function, and extreme sensitivity to DNA demethylation

3. Impaired function of primitive hematopoietic cells in mice lacking the Mixed-Lineage-Leukemia homolog Mll5

4. Mll5 contributes to hematopoietic stem cell fitness and homeostasis

5. MLL 5 protein forms intranuclear foci, and overexpression inhibits cell cycle progression

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1. The chromatin factors SET-26 and HCF-1 oppose the histone deacetylase HDA-1 in longevity and gene regulation in C. elegans;Nature Communications;2024-03-14

2. SUMO-mediated regulation of a H3K4me3 reader controls germline development inC. elegans;2024-02-28

3. Release of Histone H3K4-reading transcription factors from chromosomes in mitosis is independent of adjacent H3 phosphorylation;Nature Communications;2023-11-09

4. Defining Biological and Biochemical Functions of Noncanonical SET Domain Proteins;Journal of Molecular Biology;2023-10

5. The chromatin factors SET-26 and HCF-1 oppose the histone deacetylase HDA-1 in longevity and gene regulation inC. elegans;2023-03-23

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