APE1 is dispensable for S-region cleavage but required for its repair in class switch recombination
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference58 articles.
1. AID-dependent somatic hypermutation occurs as a DNA single-strand event in the BL2 cell line
2. Activation-Induced Cytidine Deaminase-Dependent DNA Breaks in Class Switch Recombination Occur during G1 Phase of the Cell Cycle and Depend upon Mismatch Repair
3. Specific Expression of Activation-induced Cytidine Deaminase (AID), a Novel Member of the RNA-editing Deaminase Family in Germinal Center B Cells
4. Class Switch Recombination and Hypermutation Require Activation-Induced Cytidine Deaminase (AID), a Potential RNA Editing Enzyme
5. Activation-Induced Cytidine Deaminase (AID) Deficiency Causes the Autosomal Recessive Form of the Hyper-IgM Syndrome (HIGM2)
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1. Mechanism and Regulation of Immunoglobulin Class Switch Recombination;Molecular Biology of B Cells;2024
2. Molecular Mechanism of Activation-Induced Cytidine Deaminase;Molecular Biology of B Cells;2024
3. RPA guides UNG to uracil in ssDNA to facilitate antibody class switching and repair of mutagenic uracil at the replication fork;Nucleic Acids Research;2023-11-24
4. Ago2 and a miRNA reduce Topoisomerase 1 for enhancing DNA cleavage in antibody diversification by activation-induced cytidine deaminase;Proceedings of the National Academy of Sciences;2023-04-24
5. Necessity of HuR/ELAVL1 for the activation-induced cytidine deaminase-dependent decrease in topoisomerase 1 in antibody diversification;International Immunology;2023-04-22
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