Familial hypercatabolic hypoproteinemia caused by deficiency of the neonatal Fc receptor, FcRn, due to a mutant beta2-microglobulin gene-Reference-Cited by-同舟云学术

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Familial hypercatabolic hypoproteinemia caused by deficiency of the neonatal Fc receptor, FcRn, due to a mutant beta2-microglobulin gene

Published:2006-03-20 Issue:13 Volume:103 Page:5084-5089
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Wani M. A.,Haynes L. D.,Kim J.,Bronson C. L.,Chaudhury C.,Mohanty S.,Waldmann T. A.,Robinson J. M.,Anderson C. L.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference23 articles.

1. An Fc receptor structurally related to MHC class I antigens

2. The Major Histocompatibility Complex–related Fc Receptor for IgG (FcRn) Binds Albumin and Prolongs Its Lifespan

3. Abnormally short serum half-lives of IgG in β2-microglobulin-deficient mice

4. The protection receptor for IgG catabolism is the beta2-microglobulin-containing neonatal intestinal transport receptor.

5. Increased clearance of IgG in mice that lack β 2 ‐microglobulin: possible protective role of FcRn

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