Presenilin mutations linked to familial Alzheimer's disease cause an imbalance in phosphatidylinositol 4,5-bisphosphate metabolism-Reference-Cited by-同舟云学术

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Presenilin mutations linked to familial Alzheimer's disease cause an imbalance in phosphatidylinositol 4,5-bisphosphate metabolism

Published:2006-12-08 Issue:51 Volume:103 Page:19524-19529
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Landman N.,Jeong S. Y.,Shin S. Y.,Voronov S. V.,Serban G.,Kang M. S.,Park M. K.,Di Paolo G.,Chung S.,Kim T.-W.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference49 articles.

1. Twenty Years of the Alzheimer’s Disease Amyloid Hypothesis: A Genetic Perspective

2. The Amyloid Hypothesis of Alzheimer's Disease: Progress and Problems on the Road to Therapeutics

3. The role of cerebral amyloid accumulation in common forms of Alzheimer disease

4. Take five—BACE and the γ-secretase quartet conduct Alzheimer's amyloid β-peptide generation

5. Visualization of Aβ42(43) and Aβ40 in senile plaques with end-specific Aβ monoclonals: Evidence that an initially deposited species is Aβ42(43)

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