Cadmium and Proliferation in Human Uterine Leiomyoma Cells: Evidence of a Role for EGFR/MAPK Pathways but Not Classical Estrogen Receptor Pathways-Reference-Cited by-同舟云学术

Cadmium and Proliferation in Human Uterine Leiomyoma Cells: Evidence of a Role for EGFR/MAPK Pathways but Not Classical Estrogen Receptor Pathways

Published:2015-04 Issue:4 Volume:123 Page:331-336
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environmental Health Perspectives

Author:

Gao Xiaohua¹,Yu Linda¹,Moore Alicia B.¹,Kissling Grace E.²,Waalkes Michael P.³,Dixon Darlene¹

Affiliation:

1. Molecular Pathogenesis Group, National Toxicology Program (NTP) Laboratory, Division of the NTP (DNTP)

2. Biostatistics Branch, Division of Intramural Research (DIR) and DNTP, and

3. Inorganic Toxicology Group, DNTP, National Institute of Environmental Health Sciences, National Institutes of Health, Department of Health and Human Services, Research Triangle Park, North Carolina, USA

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference50 articles.

1. Agency for Toxic Substances and Disease Registry. 2012. Toxicological Profile for Cadmium. Available: http://www.atsdr.cdc.gov/toxprofiles/tp5.pdf [accessed 10 October 2014].

2. Cadmium-Induced Effects on Cellular Signaling Pathways in the Liver of Transgenic Estrogen Reporter Mice

3. Estrogen-Like Effects of Cadmium in Vivo Do Not Appear to be Mediated via the Classical Estrogen Receptor Transcriptional Pathway

4. Cadmium Malignantly Transforms Normal Human Breast Epithelial Cells into a Basal-like Phenotype

5. Immortalization of Human Uterine Leiomyoma and Myometrial Cell Lines After Induction of Telomerase Activity: Molecular and Phenotypic Characteristics

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