Oridonin Confers Protection against Arsenic-Induced Toxicity through Activation of the Nrf2-Mediated Defensive Response-Reference-Cited by-同舟云学术

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Oridonin Confers Protection against Arsenic-Induced Toxicity through Activation of the Nrf2-Mediated Defensive Response

Published:2008-09 Issue:9 Volume:116 Page:1154-1161
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environmental Health Perspectives

Author:

Du Yu¹²,Villeneuve Nicole F.¹,Wang Xiao-Jun¹,Sun Zheng¹,Chen Weimin¹,Li Jixue¹,Lou Hongxiang²,Wong Pak Kin³,Zhang Donna D.¹

Affiliation:

1. Department of Pharmacology and Toxicology, College of Pharmacy, University of Arizona, Tucson, Arizona, USA

2. Department of Natural Products, School of Pharmaceutical Sciences, Shandong University, Shandong, People’s Republic of China

3. Aerospace and Mechanical Engineering Department, University of Arizona, Tucson, Arizona, USA

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference61 articles.

1. Accelerated DNA Adduct Formation in the Lung of the Nrf2 Knockout Mouse Exposed to Diesel Exhaust

2. Activation of Nrf2 and accumulation of ubiquitinated A170 by arsenic in osteoblasts

3. Impaired expression of glutathione synthetic enzyme genes in mice with targeted deletion of the Nrf2 basic-leucine zipper protein

4. An important function of Nrf2 in combating oxidative stress: Detoxification of acetaminophen

5. Nrf2 is essential for protection against acute pulmonary injury in mice

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