Exposure to Bisphenol A Prenatally or in Adulthood Promotes T H 2 Cytokine Production Associated with Reduction of CD4 + CD25 + Regulatory T Cells-Reference-Cited by-同舟云学术

Exposure to Bisphenol A Prenatally or in Adulthood Promotes T H 2 Cytokine Production Associated with Reduction of CD4 + CD25 + Regulatory T Cells

Published:2008-04 Issue:4 Volume:116 Page:514-519
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environmental Health Perspectives

Author:

Yan Huimin¹,Takamoto Masaya¹,Sugane Kazuo¹

Affiliation:

1. Department of Infection and Host Defense, Division of Immunology and Infectious Diseases, Shinshu University Graduate School of Medicine, Matsumoto, Japan

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference44 articles.

1. Altered allergic cytokine and antibody response in mice treated with Bisphenol A

2. The Early IL-4 Response toLeishmania majorand the Resulting Th2 Cell Maturation Steering Progressive Disease in BALB/c Mice Are Subject to the Control of Regulatory CD4+CD25+T Cells

3. The environmental pollutant pyrene induces the production of IL-4

4. Effects of perinatal exposure to bisphenol A on play behavior of female and male juvenile rats.

5. Activated CD4+ CD25+ T cells suppress antigen-specific CD4+ and CD8+ T cells but induce a suppressive phenotype only in CD4+ T cells

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