Inhibition of Ape1 nuclease activity by lead, iron, and cadmium.-Reference-Cited by-同舟云学术

Inhibition of Ape1 nuclease activity by lead, iron, and cadmium.

Published:2004-05 Issue:7 Volume:112 Page:799-804
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environmental Health Perspectives

Author:

McNeill Daniel R¹,Narayana Avinash¹,Wong Heng-Kuan¹,Wilson David M¹

Affiliation:

1. Laboratory of Molecular Gerontology, Gerontology Research Center, National Institute on Aging, Intramural Research Program, National Institutes of Health, Department of Health and Human Services, Baltimore, Maryland 21224-6825, USA.

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference45 articles.

1. Differential effects of toxic metal compounds on the activities of Fpg and XPA, two zinc finger proteins involved in DNA repair

2. Two divalent metal ions in the active site of a new crystal form of human apurinic/apyrimidinic endonuclease, ape1: implications for the catalytic mechanism 1 1Edited by I. A. Wilson

3. An exonucleolytic activity of human apurinic/apyrimidinic endonuclease on 3′ mispaired DNA

4. REPAIR OF OXIDATIVE DAMAGE TO DNA: Enzymology and Biology

5. Cloning and expression of APE, the cDNA encoding the major human apurinic endonuclease: definition of a family of DNA repair enzymes.

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