Different ATM Signaling in Response to Chromium(VI) Metabolism via Ascorbate and Nonascorbate Reduction: Implications for in Vitro Models and Toxicogenomics-Reference-Cited by-同舟云学术

Different ATM Signaling in Response to Chromium(VI) Metabolism via Ascorbate and Nonascorbate Reduction: Implications for in Vitro Models and Toxicogenomics

Published:2016-01 Issue:1 Volume:124 Page:61-66
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environ Health Perspect

Author:

Luczak Michal W.¹,Green Samantha E.¹,Zhitkovich Anatoly¹

Affiliation:

1. Department of Pathology and Laboratory Medicine, Brown University, Providence, Rhode Island, USA

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference42 articles.

1. ATSDR (Agency for Toxic Substances and Disease Registry). 2000. Toxicological Profile for Chromium. Atlanta GA ATSDR U.S. Department of Health and Human Services Available: http://www.atsdr.cdc.gov/ToxProfiles/tp7.pdf [accessed 14 November 2015].

2. Millimolar concentrations of ascorbic acid in purified human mononuclear leukocytes. Depletion and reaccumulation.;Bergsten P;J Biol Chem,1990

3. Activation of chromium(VI) by thiols results in chromium(V) formation, chromium binding to DNA and altered DNA conformation

4. Long-lived chromium(IV) and chromium(V) metabolites in the chromium(VI)-glutathione reaction: NMR, ESR, HPLC, and kinetic characterization

5. DNA Double-Strand Breaks by Cr(VI) Are Targeted to Euchromatin and Cause ATR-Dependent Phosphorylation of Histone H2AX and Its Ubiquitination

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