Exposure to dioxin and nonneoplastic mortality in the expanded IARC international cohort study of phenoxy herbicide and chlorophenol production workers and sprayers.

Author:

Vena J1,Boffetta P1,Becher H1,Benn T1,Bueno-de-Mesquita H B1,Coggon D1,Colin D1,Flesch-Janys D1,Green L1,Kauppinen T1,Littorin M1,Lynge E1,Mathews J D1,Neuberger M1,Pearce N1,Pesatori A C1,Saracci R1,Steenland K1,Kogevinas M1

Affiliation:

1. Department of Social and Preventive Medicine, State University of New York at Buffalo, USA.

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference65 articles.

1. IARC. Polychlorinated dibenzo-para-dioxins. In: IARC Monographs on the Evaluation of Carcinogenic Risks to Humans. Vol 69: Polychlorinated Dibenzo-para-dioxins and Polychlorinated Dibenzofurans. Lyon:International Agency for Research on Cancer 1997;33-343.

2. U.S. Environmental Protection Agency. Health Assessment Document for 2 3 7 8-Tetrachlorodibenzo-p-dioxin (TCDD) and Related Compounds Vols I-III. Rpt No EPA/600/BP92/001. Washington:U.S. Government Printing Office 1994.

3. Birnbaum LS. Evidence for the role of the Ah receptor in response to dioxin. In: Receptor Mediated Biological Processes: Implications for Evaluating Carcinogens. Progress in Clinical and Biological Research. Vol 387 (Spitzer HL Slaga TL Greenlee WF McClain M eds). New York:Wiley-Liss 1994;139-154.

4. The AH-receptor: genetics, structure and function

5. Xenobiotic-inducible Transcription of Cytochrome P450 Genes

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