Control of G1 arrest after DNA damage.-Reference-Cited by-同舟云学术

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Control of G1 arrest after DNA damage.

Published:1993-12 Issue:suppl 5 Volume:101 Page:55-58
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environmental Health Perspectives

Author:

Kastan M B¹,Kuerbitz S J¹

Affiliation:

1. Johns Hopkins Oncology Center, Johns Hopkins Hospital, Baltimore, MD 21205.

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference11 articles.

1. Cell Proliferation in Carcinogenesis

2. Characterization of RAD9 of Saccharomyces cerevisiae and evidence that its function acts posttranslationally in cell cycle arrest after DNA damage.

3. Participation of p53 protein in the cellular response to DNA damage;Kastan M. B.;Cancer Res.,1991

4. Suppression of Human Colorectal Carcinoma Cell Growth by Wild-Type p53

5. p53 functions as a cell cycle control protein in osteosarcomas.

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1. A Narrative Review of the <i>TP53</i> and Its Product the p53 Protein;OBM Genetics;2023-07-03

2. New RAD51 Inhibitors to Target Homologous Recombination in Human Cells;Genes;2021-06-16

3. The Chk1 inhibitor SAR-020106 sensitizes human glioblastoma cells to irradiation, to temozolomide, and to decitabine treatment;Journal of Experimental & Clinical Cancer Research;2019-10-21

4. A Temperature Sensitive Variant of p53 Drives p53-Dependent MicroRNA Expression without Evidence of Widespread Post-Transcriptional Gene Silencing;PLOS ONE;2016-02-03

5. Cytotoxicity Associated with Artemis Overexpression After Lentiviral Vector-Mediated Gene Transfer;Human Gene Therapy;2010-07

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