Effects of Wood Smoke Constituents on Mucin Gene Expression in Mice and Human Airway Epithelial Cells and on Nasal Epithelia of Subjects with a Susceptibility Gene Variant in <i>Tp53</i>-Reference-Cited by-同舟云学术

Effects of Wood Smoke Constituents on Mucin Gene Expression in Mice and Human Airway Epithelial Cells and on Nasal Epithelia of Subjects with a Susceptibility Gene Variant in Tp53

Published:2022-01 Issue:1 Volume:130 Page:
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environ Health Perspect

Author:

Tassew Dereje¹^ORCID,Fort Susan²,Mebratu Yohannes¹^ORCID,McDonald Jacob³,Chu Hong Wei⁴,Petersen Hans²^ORCID,Tesfaigzi Yohannes¹^ORCID

Affiliation:

1. Department of Medicine, Division of Pulmonary and Critical Care Medicine, Brigham and Women’s Hospital, Harvard Medical School, Boston, Massachusetts, USA

2. Chronic Obstructive Pulmonary Disease Program, Lovelace Biomedical Research Institute, Albuquerque, New Mexico, USA

3. Applied Sciences, Lovelace Biomedical Research Institute, Albuquerque, New Mexico, USA

4. Department of Medicine, National Jewish Health, Denver, Colorado, USA

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference92 articles.

1. The Presence of Chronic Mucus Hypersecretion across Adult Life in Relation to Chronic Obstructive Pulmonary Disease Development

2. SLC26A9-mediated chloride secretion prevents mucus obstruction in airway inflammation

3. Wood Smoke Enhances Cigarette Smoke–Induced Inflammation by Inducing the Aryl Hydrocarbon Receptor Repressor in Airway Epithelial Cells

4. Insulin-like growth factor-1 induces MUC8 and MUC5B expression via ERK1 and p38 MAPK in human airway epithelial cells

5. When Smoke Gets in Your Lungs

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