Genomic and proteomic profiling of responses to toxic metals in human lung cells.-Reference-Cited by-同舟云学术

Genomic and proteomic profiling of responses to toxic metals in human lung cells.

Published:2003-05 Issue:6 Volume:111 Page:825-835
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environmental Health Perspectives

Author:

Andrew Angeline S¹,Warren Amy J¹,Barchowsky Aaron¹,Temple Kaili A¹,Klei Linda¹,Soucy Nicole V¹,O'Hara Kimberley A¹,Hamilton Joshua W¹

Affiliation:

1. Department of Pharmacology and Toxicology, Dartmouth Medical School, Dartmouth College, 7927 Rubin Building, 452M-3, One Medical Center Drive, Lebanon, NH 03756-0001, USA. angeline.s.andrew@dartmouth.edu

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference56 articles.

1. Arsenic: health effects, mechanisms of actions, and research issues.

2. Chromium Toxicity and Carcinogenesis

3. Nickel-Induced Plasminogen Activator Inhibitor-1 Expression Inhibits the Fibrinolytic Activity of Human Airway Epithelial Cells

4. Decreased DNA repair gene expression among individuals exposed to arsenic in United States drinking water

5. Nickel requires hypoxia-inducible factor-1α, not redox signaling, to induce plasminogen activator inhibitor-1

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