A Systems Biology Approach Reveals Converging Molecular Mechanisms that Link Different POPs to Common Metabolic Diseases

Author:

Ruiz Patricia1,Perlina Ally2,Mumtaz Moiz1,Fowler Bruce A.3

Affiliation:

1. Computational Toxicology and Methods Development Laboratory, Division of Toxicology and Human Health Sciences, Agency for Toxic Substances and Disease Registry, Atlanta, Georgia, USA

2. Sanford Burnham Prebys Medical Discovery Institute, La Jolla, California, USA

3. Emory University Rollins School of Public Health, Atlanta, Georgia, USA

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference45 articles.

1. ATSDR (Agency for Toxic Substances and Disease Registry). 2008. Addendum to the Toxicological Profile for DDT DDE DDD. Atlanta GA ATSDRAvailable: http://www.atsdr.cdc.gov/toxprofiles/ddt_addendum.pdf [accessed 31 May 2016].

2. ATSDR. 2011. Addendum to the Toxicological Profile for Polychlorinated Biphenyls. Atlanta GA ATSDR Available: http://www.atsdr.cdc.gov/toxprofiles/pcbs_addendum.pdf [accessed 31 May 2016].

3. ATSDR. 2012. Addendum to the Toxicological Profile for Chlorinated Dibenzo-p-Dioxins (CDDs). Atlanta GA ATSDR Available: http://www.atsdr.cdc.gov/toxprofiles/cdds_addendum.pdf [accessed 31 May 2016].

4. Developmental origins of non-communicable disease: Implications for research and public health

5. POPs vs. Fat: Persistent Organic Pollutant Toxicity Targets and Is Modulated by Adipose Tissue

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