Lead Increases Lipopolysaccharide-Induced Liver Injury through Tumor Necrosis Factor-α Overexpression by Monocytes/Macrophages: Role of Protein Kinase C and p42/44 Mitogen-Activated Protein Kinase-Reference-Cited by-同舟云学术

Lead Increases Lipopolysaccharide-Induced Liver Injury through Tumor Necrosis Factor-α Overexpression by Monocytes/Macrophages: Role of Protein Kinase C and p42/44 Mitogen-Activated Protein Kinase

Published:2006-04 Issue:4 Volume:114 Page:507-513
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environmental Health Perspectives

Author:

Cheng Yu-Jung¹²,Yang Bei-Chang²³,Liu Ming-Yie¹²

Affiliation:

1. Department of Environmental and Occupational Health,

2. Institute of Basic Medical Sciences and

3. Department of Microbiology and Immunology, National Cheng Kung University Medical College, Tainan, Taiwan

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference46 articles.

1. Signalling pathways of the TNF superfamily: a double-edged sword

2. The Biology of Cachectin/TNF -- A Primary Mediator of the Host Response

3. Pharmacokinetic model to describe the disposition of lead in the rat

4. MODULATION OF TUMOR NECROSIS FACTOR-?? AND OXIDATIVE STRESS THROUGH PROTEIN KINASE C AND P42/44 MITOGEN-ACTIVATED PROTEIN KINASE IN LEAD INCREASES LIPOPOLYSACCHARIDE-INDUCED LIVER DAMAGE IN RATS

5. Regulation of tumor necrosis factor-alpha in glioma cells by lead and lipopoly-saccharide: involvement of common signaling pathway;Cheng YJ;Toxicol Lett,2004

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