IL-33 Drives Augmented Responses to Ozone in Obese Mice-Reference-Cited by-同舟云学术

IL-33 Drives Augmented Responses to Ozone in Obese Mice

Published:2017-02 Issue:2 Volume:125 Page:246-253
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environmental Health Perspectives

Author:

Mathews Joel A.¹,Krishnamoorthy Nandini²,Kasahara David Itiro¹,Cho Youngji¹,Wurmbrand Allison Patricia¹,Ribeiro Luiza¹,Smith Dirk³,Umetsu Dale⁴,Levy Bruce D.²,Shore Stephanie Ann¹

Affiliation:

1. Department of Environmental Health, Harvard T.H. Chan School of Public Health, Boston, Massachusetts, USA

2. Pulmonary and Critical Care Medicine, Harvard Institutes of Medicine Building, Boston, Massachusetts, USA

3. Department of Inflammation Research, Amgen, Seattle, Washington, USA

4. Genentech, South San Francisco, California, USA

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference38 articles.

1. Ozone Exposure and Lung Function

2. IL-33 is more potent than IL-25 in provoking IL-13–producing nuocytes (type 2 innate lymphoid cells) and airway contraction

3. Acute Pulmonary Function Response to Ozone in Young Adults As a Function of Body Mass Index

4. IL-33: an alarmin cytokine with crucial roles in innate immunity, inflammation and allergy

5. The role of type 2 innate lymphoid cells in asthma

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