Fluticasone Propionate Protects against Ozone-Induced Airway Inflammation and Modified Immune Cell Activation Markers in Healthy Volunteers-Reference-Cited by-同舟云学术

Fluticasone Propionate Protects against Ozone-Induced Airway Inflammation and Modified Immune Cell Activation Markers in Healthy Volunteers

Published:2008-06 Issue:6 Volume:116 Page:799-805
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environmental Health Perspectives

Author:

Alexis Neil E.¹²,Lay John C.¹,Haczku Angela³,Gong Henry⁴⁵,Linn William⁴⁵,Hazucha Milan J.¹,Harris Brad¹,Tal-Singer Ruth⁶,Peden David B.¹²

Affiliation:

1. Center for Environmental Medicine, Asthma and Lung Biology, and

2. Department of Medicine, University of North Carolina, Chapel Hill, North Carolina, USA

3. Department of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, USA

4. Department of Medicine, University of California, Los Angeles, California, USA

5. Environmental Health Service, Rancho Los Amigos National Rehabilitation Center, Downey, California, USA

6. GlaxoSmithKline, King of Prussia, Pennsylvania, USA

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference37 articles.

1. Circulating CD11b expression correlates with the neutrophil response and airway mCD14 expression is enhanced following ozone exposure in humans

2. Effect of inhaled endotoxin on airway and circulating inflammatory cell phagocytosis and CD11b expression in atopic asthmatic subjects

3. Effect of inhaled endotoxin on airway and circulating inflammatory cell phagocytosis and CD11b expression in atopic asthmatic subjects;Alexis NE;J Allergy Clin Immunol,2004

4. Biological material on inhaled coarse fraction particulate matter activates airway phagocytes in vivo in healthy volunteers

5. Blunting airway eosinophilic inflammation results in a decreased airway neutrophil response to inhaled LPS in patients with atopic asthma: A role for CD14

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