Tumor Suppressor Gene Inactivation during Cadmium-Induced Malignant Transformation of Human Prostate Cells Correlates with Overexpression of de Novo DNA Methyltransferase

Author:

Benbrahim-Tallaa Lamia1,Waterland Robert A.2,Dill Anna L.1,Webber Mukta M.3,Waalkes Michael P.1

Affiliation:

1. Inorganic Carcinogenesis Section, Laboratory of Comparative Carcinogenesis, National Cancer Institute at the National Institute of Environmental Health Sciences, National Institutes of Health, Department of Health and Human Services, Research Triangle Park, North Carolina, USA

2. Departments of Pediatrics and Molecular and Human Genetics, Baylor College of Medicine, Children’s Nutrition Research Center, Houston, Texas, USA

3. Department of Medicine and Department of Zoology, Michigan State University, East Lansing, Michigan, USA

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference32 articles.

1. Cadmium-induced malignant transformation of human prostate epithelial cells;Achanzar WE;Cancer Res,2001

2. Methylation of tumor suppressor genes p16(INK4a), p27(Kip1) and E-cadherin in carcinogenesis

3. DNA Methylation in Folate Deficiency: Use of CpG Methylase

4. Alterations in DNA Methylation: A Fundamental Aspect of Neoplasia

5. Abnormal patterns of DNA methylation in human neoplasia: potential consequences for tumor progression;Baylin SB;Cancer Cells,1991

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