Adipose-specific disruption of autotaxin enhances nutritional fattening and reduces plasma lysophosphatidic acid-Reference-Cited by-同舟云学术

Adipose-specific disruption of autotaxin enhances nutritional fattening and reduces plasma lysophosphatidic acid

Published:2011-06 Issue:6 Volume:52 Page:1247-1255
ISSN:0022-2275
Container-title:Journal of Lipid Research
language:en
Short-container-title:Journal of Lipid Research

Author:

Dusaulcy Rodolphe,Rancoule Chloé,Grès Sandra,Wanecq Estelle,Colom André,Guigné Charlotte,van Meeteren Laurens A.,Moolenaar Wouter H.,Valet Philippe,Saulnier-Blache Jean Sébastien

Publisher

Elsevier BV

Subject

Cell Biology,Endocrinology,Biochemistry

Reference22 articles.

1. Regulation and biological activities of the autotaxin-LPA axis;van Meeteren;Prog. Lipid Res.,2007

2. Autotaxin (ATX): a multi-functional and multi-modular protein possessing enzymatic lysoPLD activity and matricellular properties;Yuelling;Biochim. Biophys. Acta.,2008

3. Autotaxin;Boutin;Cell. Mol. Life Sci.,2009

4. Autotaxin, a secreted lysophospholipase D, is essential for blood vessel formation during development;van Meeteren;Mol. Cell. Biol.,2006

5. Autotaxin stabilizes blood vessels and is required for embryonic vasculature by producing lysophosphatidic acid;Tanaka;J. Biol. Chem.,2006

Cited by 160 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Autotaxin–Lysophosphatidate Axis: Promoter of Cancer Development and Possible Therapeutic Implications;International Journal of Molecular Sciences;2024-07-15

2. Lysophosphatidic acid metabolism and signaling in heart disease;Canadian Journal of Physiology and Pharmacology;2024-07-05

3. Renal tubule-specific Atgl deletion links kidney lipid metabolism to glucagon-like peptide 1 and insulin secretion independent of renal inflammation or lipotoxicity;Molecular Metabolism;2024-03

4. Autotaxin and Lysophosphatidate Signaling: Prime Targets for Mitigating Therapy Resistance in Breast Cancer;World Journal of Oncology;2024-02

5. Bioactive signalling lipids as drivers of chronic liver diseases;Journal of Hepatology;2024-01