Macrophage deficiency of Akt2 reduces atherosclerosis in Ldlr null mice-Reference-Cited by-同舟云学术

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Macrophage deficiency of Akt2 reduces atherosclerosis in Ldlr null mice

Published:2014-11 Issue:11 Volume:55 Page:2296-2308
ISSN:0022-2275
Container-title:Journal of Lipid Research
language:en
Short-container-title:Journal of Lipid Research

Author:

Babaev Vladimir R.,Hebron Katie E.,Wiese Carrie B.,Toth Cynthia L.,Ding Lei,Zhang Youmin,May James M.,Fazio Sergio,Vickers Kasey C.,Linton MacRae F.

Publisher

Elsevier BV

Subject

Cell Biology,Endocrinology,Biochemistry

Reference59 articles.

1. Macrophages in atherosclerosis: a dynamic balance;Moore;Nat. Rev. Immunol.,2013

2. AKT/PKB signaling: navigating downstream;Manning;Cell.,2007

3. Constitutively activated Akt-1 is vital for the survival of human monocyte-differentiated macrophages. Role of Mcl-1, independent of nuclear factor (NF)-kappaB, Bad, or caspase activation;Liu;J. Exp. Med.,2001

4. Growth retardation and increased apoptosis in mice with homozygous disruption of the Akt1 gene;Chen;Genes Dev.,2001

5. Akt1/PKBalpha is required for normal growth but dispensable for maintenance of glucose homeostasis in mice;Cho;J. Biol. Chem.,2001

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