Hepatic insulin resistance in ob/ob mice involves increases in ceramide, aPKC activity, and selective impairment of Akt-dependent FoxO1 phosphorylation-Reference-Cited by-同舟云学术

Hepatic insulin resistance in ob/ob mice involves increases in ceramide, aPKC activity, and selective impairment of Akt-dependent FoxO1 phosphorylation

Published:2015-01 Issue:1 Volume:56 Page:70-80
ISSN:0022-2275
Container-title:Journal of Lipid Research
language:en
Short-container-title:Journal of Lipid Research

Author:

Sajan Mini P.,Ivey Robert A.,Lee Mackenzie C.,Farese Robert V.

Publisher

Elsevier BV

Subject

Cell Biology,Endocrinology,Biochemistry

Reference29 articles.

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2. Muscle-specific knockout of PKC-λ impairs glucose transport and induces metabolic and diabetic syndromes;Farese;J. Clin. Invest.,2007

3. Insulin-induced Activation of Atypical Protein Kinase C, but Not Protein Kinase B, Is Maintained in Diabetic (ob/ob and Goto-Kakazaki) Liver

4. Critical role of atypical protein kinase C in activating hepatic SREBP-1c and NFκB in obesity;Sajan;J. Lipid Res.,2009

5. Akt-Dependent Phosphorylation of Hepatic FoxO1 Is Compartmentalized on a WD40/ProF Scaffold and Is Selectively Inhibited by aPKC in Early Phases of Diet-Induced Obesity

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