CD36, but not G2A, modulates efferocytosis, inflammation, and fibrosis following bleomycin-induced lung injury

Author:

Parks Brian W.,Black Leland L.,Zimmerman Kurt A.,Metz Allison E.,Steele Chad,Murphy-Ullrich Joanne E.,Kabarowski Janusz H.

Publisher

Elsevier BV

Subject

Cell Biology,Endocrinology,Biochemistry

Reference59 articles.

1. Apoptotic cell removal in development and tissue homeostasis;Henson;Trends Immunol.,2006

2. Engulfment of apoptotic cells: signals for a good meal;Ravichandran;Nat. Rev. Immunol.,2007

3. CD36 is required for phagocytosis of apoptotic cells by human macrophages that use either a phosphatidylserine receptor or the vitronectin receptor (alpha v beta 3);Fadok;J. Immunol.,1998

4. Cell death, remodeling, and repair in chronic obstructive pulmonary disease?;Henson;Proc. Am. Thorac. Soc.,2006

5. Identification of a novel family of oxidized phospholipids that serve as ligands for the macrophage scavenger receptor CD36;Podrez;J. Biol. Chem.,2002

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