Abstract
SUMMARYCells carryingtraJ−mutants of F are transfer-deficient and are good recipients in conjugation (Achtman, Willets & Clark, 1972). In addition, thetraJgene product is involved in pilus- and in plasmid-specificity (Willetts, 1971). J-independent mutants were isolated as revertants of atraJ−mutant; they still carry thetraJ−mutation but also carry at least one other mutation which results in transfer in the absence of thetraJgene product. J-independent transfer of these mutants is not inhibited by the R 100 repressor. Various models are presented which can account for the properties oftraJ−mutants and of these J-independent revertants.
Subject
Genetics,General Medicine
Cited by
19 articles.
订阅此论文施引文献
订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献