Disturbed Flow Enhances Inflammatory Signaling and Atherogenesis by Increasing Thioredoxin-1 Level in Endothelial Cell Nuclei-Reference-Cited by-同舟云学术

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Disturbed Flow Enhances Inflammatory Signaling and Atherogenesis by Increasing Thioredoxin-1 Level in Endothelial Cell Nuclei

Published:2014-09-29 Issue:9 Volume:9 Page:e108346
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Go Young-Mi,Son Dong Ju,Park Heonyong,Orr Michael,Hao Li,Takabe Wakako,Kumar Sandeep,Kang Dong Won,Kim Chan Woo,Jo Hanjoong,Jones Dean P.

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference59 articles.

1. Effects of shear stress and stretch on endothelial function;J Ando;Antioxidants & redox signaling,2011

2. Hemodynamics and the focal origin of atherosclerosis: a spatial approach to endothelial structure, gene expression, and function;PF Davies;Annals of the New York Academy of Sciences,2001

3. Animal, in vitro, and ex vivo models of flow-dependent atherosclerosis: role of oxidative stress;A Rezvan;Antioxidants & redox signaling,2011

4. Effects of disturbed flow on vascular endothelium: pathophysiological basis and clinical perspectives;JJ Chiu;Physiological reviews,2011

5. Bone morphogenic protein 4 produced in endothelial cells by oscillatory shear stress induces monocyte adhesion by stimulating reactive oxygen species production from a nox1-based NADPH oxidase;GP Sorescu;Circulation research,2004

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