The GIP Receptor Displays Higher Basal Activity than the GLP-1 Receptor but Does Not Recruit GRK2 or Arrestin3 Effectively-Reference-Cited by-同舟云学术

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The GIP Receptor Displays Higher Basal Activity than the GLP-1 Receptor but Does Not Recruit GRK2 or Arrestin3 Effectively

Published:2014-09-05 Issue:9 Volume:9 Page:e106890
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Al-Sabah Suleiman,Al-Fulaij Munya,Shaaban Ghina,Ahmed Hanadi A.,Mann Rosalind J.,Donnelly Dan,Bünemann Moritz,Krasel Cornelius

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference37 articles.

1. Biology of incretins: GLP-1 and GIP;LL Baggio;Gastroenterology,2007

2. The physiology of glucagon-like peptide 1;JJ Holst;Physiol Rev,2007

3. Loss of incretin effect is a specific, important, and early characteristic of type 2 diabetes;JJ Holst;Diabetes Care,2011

4. The future of incretin-based therapy: novel avenues–novel targets;B Ahren;Diabetes Obes Metab,2011

5. Preserved incretin activity of glucagon-like peptide 1 [7-36 amide] but not of synthetic human gastric inhibitory polypeptide in patients with type-2 diabetes mellitus;MA Nauck;J Clin Invest,1993

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1. GIP-derived GIP receptor antagonists – a review of their role in GIP receptor pharmacology;Peptides;2024-07

2. Reduced incretin receptor trafficking upon activation enhances glycemic control and reverses obesity in diet-induced obese mice;American Journal of Physiology-Cell Physiology;2024-07-01

3. Lipid regulation of the glucagon receptor family;Journal of Endocrinology;2024-04-10

4. GLP-1 and GIP receptors signal through distinct β-arrestin 2-dependent pathways to regulate pancreatic β cell function;Cell Reports;2023-11

5. Enhanced Endosomal Signaling and Desensitization of GLP-1R vs GIPR in Pancreatic Beta Cells;Endocrinology;2023-02-12

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