Complex I Deficiency Due to Selective Loss of Ndufs4 in the Mouse Heart Results in Severe Hypertrophic Cardiomyopathy-Reference-Cited by-同舟云学术

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Complex I Deficiency Due to Selective Loss of Ndufs4 in the Mouse Heart Results in Severe Hypertrophic Cardiomyopathy

Published:2014-04-04 Issue:4 Volume:9 Page:e94157
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Chouchani Edward T.,Methner Carmen,Buonincontri Guido,Hu Chou-Hui,Logan Angela,Sawiak Stephen J.,Murphy Michael P.,Krieg Thomas

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference32 articles.

1. The role of mitochondria in the salvage and the injury of the ischemic myocardium;F Di Lisa;Biochim Biophys Acta,1998

2. Mitochondrial permeability transition pore opening during myocardial reperfusion—a target for cardioprotection;AP Halestrap;Cardiovascular research,2004

3. Mouse models of mitochondrial dysfunction and heart failure;LK Russell;J Mol Cell Cardiol,2005

4. Mitochondria play a critical role in cardioprotection;J Marin-Garcia;J Card Fail,2004

5. Mitochondrial complex I deficiency leads to increased production of superoxide radicals and induction of superoxide dismutase;S Pitkanen;J Clin Invest,1996

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