On the Role of the Gap Junction Protein Cx43 (GJA1) in Human Cardiac Malformations with Fallot-Pathology. A Study on Paediatric Cardiac Specimen-Reference-Cited by-同舟云学术

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On the Role of the Gap Junction Protein Cx43 (GJA1) in Human Cardiac Malformations with Fallot-Pathology. A Study on Paediatric Cardiac Specimen

Published:2014-04-21 Issue:4 Volume:9 Page:e95344
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Salameh Aida,Haunschild Josphina,Bräuchle Paul,Peim Oliver,Seidel Thomas,Reitmann Marko,Kostelka Martin,Bakhtiary Farhad,Dhein Stefan,Dähnert Ingo

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference38 articles.

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2. Mutations of the Connexin43 gap-junction gene in patients with heart malformations and defects of laterality;SH Britz-Cunningham;N Engl J Med,1995

3. Evaluating the role of connexin43 in congenital heart disease: Screening for mutations in patients with outflow tract anomalies and the analysis of knock-in mouse models;GY Huang;J Cardiovasc Dis Res,2011

4. Altered patterns of cardiac intercellular junction distribution in hypertrophic cardiomyopathy;R Sepp;Heart,1996

5. The signal transduction cascade regulating the expression of the gap junction protein connexin43 by β-adrenoceptors;A Salameh;Br J Pharmacol,2009

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