Pathogenic VCP/TER94 Alleles Are Dominant Actives and Contribute to Neurodegeneration by Altering Cellular ATP Level in a Drosophila IBMPFD Model-Reference-Cited by-同舟云学术

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Pathogenic VCP/TER94 Alleles Are Dominant Actives and Contribute to Neurodegeneration by Altering Cellular ATP Level in a Drosophila IBMPFD Model

Published:2011-02-03 Issue:2 Volume:7 Page:e1001288
ISSN:1553-7404
Container-title:PLoS Genetics
language:en
Short-container-title:PLoS Genet

Author:

Chang Ya-Chu,Hung Wan-Tzu,Chang Yun-Chin,Chang Henry C.,Wu Chia-Lin,Chiang Ann-Shyn,Jackson George R.,Sang Tzu-Kang

Publisher

Public Library of Science (PLoS)

Subject

Cancer Research,Genetics (clinical),Genetics,Molecular Biology,Ecology, Evolution, Behavior and Systematics

Reference60 articles.

1. Inclusion body myopathy associated with Paget disease of bone and frontotemporal dementia is caused by mutant valosin-containing protein.;GD Watts;Nat Genet,2004

2. Valosin-containing protein is a multi-ubiquitin chain-targeting factor required in ubiquitin-proteasome degradation.;RM Dai;Nat Cell Biol,2001

3. Protein dislocation from the ER requires polyubiquitination and the AAA-ATPase Cdc48.;E Jarosch;Nat Cell Biol,2002

4. Membrane fusion and the cell cycle: Cdc48p participates in the fusion of ER membranes.;M Latterich;Cell,1995

5. An NSF-like ATPase, p97, and NSF mediate cisternal regrowth from mitotic Golgi fragments.;C Rabouille;Cell,1995

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