DNA replication stress triggers rapid DNA replication fork breakage by Artemis and XPF-Reference-Cited by-同舟云学术

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DNA replication stress triggers rapid DNA replication fork breakage by Artemis and XPF

Published:2018-07-30 Issue:7 Volume:14 Page:e1007541
ISSN:1553-7404
Container-title:PLOS Genetics
language:en
Short-container-title:PLoS Genet

Author:

Bétous Rémy^ORCID,Goullet de Rugy Théo,Pelegrini Alessandra Luiza,Queille Sophie,de Villartay Jean-Pierre^ORCID,Hoffmann Jean-Sébastien^ORCID

Funder

Agence Nationale de la Recherche

Ligue Nationale Contre le Cancer

Institut National Du Cancer

Fondation de France

Fundação de Amparo à Pesquisa do Estado de São Paulo

Publisher

Public Library of Science (PLoS)

Subject

Cancer Research,Genetics (clinical),Genetics,Molecular Biology,Ecology, Evolution, Behavior and Systematics

Reference29 articles.

1. Causes and consequences of replication stress;MK Zeman;Nat Cell Biol,2014

2. DNA replication stress as a hallmark of cancer;M Macheret;Annu Rev Pathol,2015

3. The essential kinase ATR: ensuring faithful duplication of a challenging genome;JC Saldivar;Nat Rev Mol Cell Biol,2017

4. Hydroxyurea-stalled replication forks become progressively inactivated and require two different RAD51-mediated pathways for restart and repair;E Petermann;Mol Cell,2010

5. The structure-specific endonuclease Mus81 contributes to replication restart by generating double-strand DNA breaks;K Hanada;Nat Struct Mol Biol,2007

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3. ATR limits Rad18-mediated PCNA monoubiquitination to preserve replication fork and telomerase-independent telomere stability;The EMBO Journal;2024-03-11

4. RAD51 restricts DNA over-replication from re-activated origins;The EMBO Journal;2024-02-15

5. Dclre1c-Mutation-Induced Immunocompromised Mice Are a Novel Model for Human Xenograft Research;Biomolecules;2024-02-02

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