Germline mutations and somatic inactivation of TRIM28 in Wilms tumour

Author:

Halliday Benjamin J.,Fukuzawa RyujiORCID,Markie David M.,Grundy Richard G.,Ludgate Jackie L.,Black Michael A.,Skeen Jane E.ORCID,Weeks Robert J.,Catchpoole Daniel R.,Roberts Aedan G. K.ORCID,Reeve Anthony E.,Morison Ian M.ORCID

Funder

Ministry of Business, Innovation and Employment (NZ)

Japanese Society for the Promotion of Science

Cure Kids

University of Otago Dunedin School of Medicine

Maurice and Phyllis Paykel Trust (NZ)

Tokyo Metropolitan Government

Publisher

Public Library of Science (PLoS)

Subject

Cancer Research,Genetics(clinical),Genetics,Molecular Biology,Ecology, Evolution, Behavior and Systematics

Reference56 articles.

1. Epidemiology of Wilms tumor;N Breslow;Med Pediatr Oncol,1993

2. An internal deletion within an 11p13 zinc finger gene contributes to the development of Wilms' tumor;DA Haber;Cell,1990

3. WT-1 is required for early kidney development;JA Kreidberg;Cell,1993

4. The Role of WT1 in Embryonic Development and Normal Organ Homeostasis;B Wilm;Methods Mol Biol,2016

5. Mutational activation of the beta-catenin proto-oncogene is a common event in the development of Wilms' tumors;R Koesters;Cancer Res,1999

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