GLIS3, a Susceptibility Gene for Type 1 and Type 2 Diabetes, Modulates Pancreatic Beta Cell Apoptosis via Regulation of a Splice Variant of the BH3-Only Protein Bim-Reference-Cited by-同舟云学术

GLIS3, a Susceptibility Gene for Type 1 and Type 2 Diabetes, Modulates Pancreatic Beta Cell Apoptosis via Regulation of a Splice Variant of the BH3-Only Protein Bim

Published:2013-05-30 Issue:5 Volume:9 Page:e1003532
ISSN:1553-7404
Container-title:PLoS Genetics
language:en
Short-container-title:PLoS Genet

Author:

Nogueira Tatiane C.,Paula Flavia M.,Villate Olatz,Colli Maikel L.,Moura Rodrigo F.,Cunha Daniel A.,Marselli Lorella,Marchetti Piero,Cnop Miriam,Julier Cécile,Eizirik Decio L.

Publisher

Public Library of Science (PLoS)

Subject

Cancer Research,Genetics(clinical),Genetics,Molecular Biology,Ecology, Evolution, Behavior and Systematics

Reference74 articles.

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2. Novel GLIS3 mutations demonstrate an extended multisystem phenotype;P Dimitri;Eur J Endocrinol,2011

3. Genome-wide association study and meta-analysis find that over 40 loci affect risk of type 1 diabetes;JC Barrett;Nat Genet,2009

4. New genetic loci implicated in fasting glucose homeostasis and their impact on type 2 diabetes risk;J Dupuis;Nat Genet,2010

5. Variants at DGKB/TMEM195, ADRA2A, GLIS3 and C2CD4B loci are associated with reduced glucose-stimulated β-cell function in middle-aged Danish people;TW Boesgaard;Diabetologia,2010

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