Congenic Mice Provide Evidence for a Genetic Locus That Modulates Spontaneous Arthritis Caused by Deficiency of IL-1RA
Author:
Publisher
Public Library of Science (PLoS)
Subject
Multidisciplinary
Reference18 articles.
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3. An autoinflammatory disease with deficiency of the interleukin-1-receptor antagonist;I Aksentijevich;N Engl J Med,2009
4. Osteoporotic fractures are associated with an 86-base pair repeat polymorphism in the interleukin-1–receptor antagonist gene but not with polymorphisms in the interleukin-1beta gene;BL Langdahl;J Bone Miner Res,2000
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Cited by 5 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. A New Understanding of the Role of IL‐1 in Age‐Related Intervertebral Disc Degeneration in a Murine Model;Journal of Bone and Mineral Research;2019-05-29
2. Decreased expression levels of Ifi genes is associated to the increased resistance to spontaneous arthritis disease in mice deficiency of IL-1RA;BMC Immunology;2016-08-02
3. Validation of Simple Sequence Length Polymorphism Regions of Commonly Used Mouse Strains for Marker Assisted Speed Congenics Screening;International Journal of Genomics;2015
4. Genomic locus on chromosome 1 regulates susceptibility to spontaneous arthritis in mice deficiency of IL-1RA;BMC Immunology;2014-12
5. Analysis of effect of congenic mice on the gene expression under genomic background of BALB/c mice;BMC Bioinformatics;2014-09-29
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