A Bispecific Protein Capable of Engaging CTLA-4 and MHCII Protects Non-Obese Diabetic Mice from Autoimmune Diabetes-Reference-Cited by-同舟云学术

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A Bispecific Protein Capable of Engaging CTLA-4 and MHCII Protects Non-Obese Diabetic Mice from Autoimmune Diabetes

Published:2013-05-21 Issue:5 Volume:8 Page:e63530
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Zhao Hongmei,Karman Jozsef,Jiang Ji-Lei,Zhang Jinhua,Gumlaw Nathan,Lydon John,Zhou Qun,Qiu Huawei,Jiang Canwen,Cheng Seng H.,Zhu Yunxiang

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference66 articles.

1. CTLA-4: a negative regulator of autoimmune disease;NJ Karandikar;J Exp Med,1996

2. CD28 and CTLA-4 have opposing effects on the response of T cells to stimulation;MF Krummel;J Exp Med,1995

3. Lymphocyte activation: T-cell regulation by CTLA-4;PS Linsley;Curr Biol,1996

4. CTLA-4 regulates tolerance induction and T cell differentiation in vivo;TL Walunas;J Immunol,1998

5. CTLA-4 can function as a negative regulator of T cell activation;TL Walunas;Immunity,1994

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1. From bench to bedside: targeting lymphocyte activation gene 3 as a therapeutic strategy for autoimmune diseases;Inflammation Research;2023-06

2. The Mechanistic Impact of N-Glycosylation on Stability, Pharmacokinetics, and Immunogenicity of Therapeutic Proteins;Journal of Pharmaceutical Sciences;2019-04

3. Bispecific antibodies: The next generation of targeted inflammatory bowel disease therapies;Autoimmunity Reviews;2019-02

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