The Endogenous Th17 Response in NO2-Promoted Allergic Airway Disease Is Dispensable for Airway Hyperresponsiveness and Distinct from Th17 Adoptive Transfer

Author:

Martin Rebecca A.,Ather Jennifer L.,Daggett Rebecca,Hoyt Laura,Alcorn John F.,Suratt Benjamin T.,Weiss Daniel J.,Lundblad Lennart K. A.,Poynter Matthew E.

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

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5. Airway epithelial regulation of allergic sensitization in asthma;ME Poynter;Pulm Pharmacol Ther,2012

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